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diffuse large b cell lymphoma sudhl4  (DSMZ)


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    DSMZ diffuse large b cell lymphoma sudhl4
    Diffuse Large B Cell Lymphoma Sudhl4, supplied by DSMZ, used in various techniques. Bioz Stars score: 95/100, based on 123 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/diffuse+large+b+cell+lymphoma+sudhl4/WSU-DLCL2/pm38934080-69-26-32
    Average 95 stars, based on 123 article reviews
    diffuse large b cell lymphoma sudhl4 - by Bioz Stars, 2026-09
    95/100 stars

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    other:

    Article Title: Core regions in immunoglobulin heavy chain enhancers essential for survival of non-Hodgkin lymphoma cells are identified by a CRISPR interference screen.
    Article Snippet: Burkitt lymphoma: BL41, CA46, DG75 (DSMZ, Braunschweig, Germany), ST486 (ATCC, LGC Standards, Lomianki, Poland), BL2, BL60, JI, LY19 (gift from prof. Reiner Siebert, Ulm University, Germany), diffuse large B-cell lymphoma: SUDHL4, WSU-DLCL2 (DSMZ) and P493-6 17 (gift from prof. D. Eick, Helmholtz Center, Munich, Germany) were grown in RPMI 1640 (Lonza, Basel, Switzerland) and HEK293T (DSMZ) in DMEM (Lonza), supplemented with 2mM L-glutamine, 1% Penicillin-Streptomycin (Biowest, Nuaillé, France) and 10-20% fetal bovine serum (Sigma-Aldrich, Saint Louis, MO, USA).



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    diffuse large b cell lymphoma cell lines sudhl4  (ATCC)
    96
    ATCC diffuse large b cell lymphoma cell lines sudhl4
    Establishment of Epstein‐Barr virus (EBV)‐infected diffuse large B‐cell lymphoma (DLBCL) cell lines. A) Schematic illustration showing the process of establishing EBV‐positive DLBCL cell lines. EBV‐negative DLBCL cell lines <t>(SUDHL4</t> and SUDHL6) were infected with cell‐free EBV derived from Akata cells and subsequently selected using G418. This figure was created by Figdraw under copyright code‐[RURPI77371]. B) Quantification of latency gene expression in different cell lines by quantitative reverse transcription PCR (qRT‐PCR), including EBER1, EBER2, LMP1, LMP2A, EBNA1, EBNA2, EBNA3A, EBNA3B, and EBNA3C. Gene expression was normalized to β‐actin. C) Western blot analysis confirms the expression of EBNA2 and LMP1 in the indicated cell lines. Akata cells were stimulated with 0.8% goat anti‐human IgG for 6 h. D) Mapping analysis of transcript sequences from tumors derived from SUDHL6‐EBV or SUDHL6 cells against the Akata genome. E) Assessment of latency genes expression in xenograft tumors from EBV positive DLBCL cells and their parental counterparts. F) Representative images of EBERs‐ISH in tumor tissues from mice implanted with EBV‐positive DLBCL cells or their parental cells.
    Diffuse Large B Cell Lymphoma Cell Lines Sudhl4, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/diffuse+large+b+cell+lymphoma+sudhl4/SU-DHL-4/pmc12412552-196-2-16
    Average 96 stars, based on 1 article reviews
    diffuse large b cell lymphoma cell lines sudhl4 - by Bioz Stars, 2026-09
    96/100 stars
    		  Buy from Supplier
    diffuse large b cell lymphoma sudhl4  (DSMZ)
    95
    DSMZ diffuse large b cell lymphoma sudhl4
    Establishment of Epstein‐Barr virus (EBV)‐infected diffuse large B‐cell lymphoma (DLBCL) cell lines. A) Schematic illustration showing the process of establishing EBV‐positive DLBCL cell lines. EBV‐negative DLBCL cell lines <t>(SUDHL4</t> and SUDHL6) were infected with cell‐free EBV derived from Akata cells and subsequently selected using G418. This figure was created by Figdraw under copyright code‐[RURPI77371]. B) Quantification of latency gene expression in different cell lines by quantitative reverse transcription PCR (qRT‐PCR), including EBER1, EBER2, LMP1, LMP2A, EBNA1, EBNA2, EBNA3A, EBNA3B, and EBNA3C. Gene expression was normalized to β‐actin. C) Western blot analysis confirms the expression of EBNA2 and LMP1 in the indicated cell lines. Akata cells were stimulated with 0.8% goat anti‐human IgG for 6 h. D) Mapping analysis of transcript sequences from tumors derived from SUDHL6‐EBV or SUDHL6 cells against the Akata genome. E) Assessment of latency genes expression in xenograft tumors from EBV positive DLBCL cells and their parental counterparts. F) Representative images of EBERs‐ISH in tumor tissues from mice implanted with EBV‐positive DLBCL cells or their parental cells.
    Diffuse Large B Cell Lymphoma Sudhl4, supplied by DSMZ, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/diffuse+large+b+cell+lymphoma+sudhl4/WSU-DLCL2/pm38934080-69-26-32
    Average 95 stars, based on 1 article reviews
    diffuse large b cell lymphoma sudhl4 - by Bioz Stars, 2026-09
    95/100 stars
    		  Buy from Supplier

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    Establishment of Epstein‐Barr virus (EBV)‐infected diffuse large B‐cell lymphoma (DLBCL) cell lines. A) Schematic illustration showing the process of establishing EBV‐positive DLBCL cell lines. EBV‐negative DLBCL cell lines (SUDHL4 and SUDHL6) were infected with cell‐free EBV derived from Akata cells and subsequently selected using G418. This figure was created by Figdraw under copyright code‐[RURPI77371]. B) Quantification of latency gene expression in different cell lines by quantitative reverse transcription PCR (qRT‐PCR), including EBER1, EBER2, LMP1, LMP2A, EBNA1, EBNA2, EBNA3A, EBNA3B, and EBNA3C. Gene expression was normalized to β‐actin. C) Western blot analysis confirms the expression of EBNA2 and LMP1 in the indicated cell lines. Akata cells were stimulated with 0.8% goat anti‐human IgG for 6 h. D) Mapping analysis of transcript sequences from tumors derived from SUDHL6‐EBV or SUDHL6 cells against the Akata genome. E) Assessment of latency genes expression in xenograft tumors from EBV positive DLBCL cells and their parental counterparts. F) Representative images of EBERs‐ISH in tumor tissues from mice implanted with EBV‐positive DLBCL cells or their parental cells.

    Journal: Advanced Science

    Article Title: Tumor‐Associated Sympathetic Nerves Promote the Progression of Epstein‐Barr Virus‐Positive Diffuse Large B‐Cell Lymphoma

    doi: 10.1002/advs.202413580

    Figure Lengend Snippet: Establishment of Epstein‐Barr virus (EBV)‐infected diffuse large B‐cell lymphoma (DLBCL) cell lines. A) Schematic illustration showing the process of establishing EBV‐positive DLBCL cell lines. EBV‐negative DLBCL cell lines (SUDHL4 and SUDHL6) were infected with cell‐free EBV derived from Akata cells and subsequently selected using G418. This figure was created by Figdraw under copyright code‐[RURPI77371]. B) Quantification of latency gene expression in different cell lines by quantitative reverse transcription PCR (qRT‐PCR), including EBER1, EBER2, LMP1, LMP2A, EBNA1, EBNA2, EBNA3A, EBNA3B, and EBNA3C. Gene expression was normalized to β‐actin. C) Western blot analysis confirms the expression of EBNA2 and LMP1 in the indicated cell lines. Akata cells were stimulated with 0.8% goat anti‐human IgG for 6 h. D) Mapping analysis of transcript sequences from tumors derived from SUDHL6‐EBV or SUDHL6 cells against the Akata genome. E) Assessment of latency genes expression in xenograft tumors from EBV positive DLBCL cells and their parental counterparts. F) Representative images of EBERs‐ISH in tumor tissues from mice implanted with EBV‐positive DLBCL cells or their parental cells.

    Article Snippet: The human diffuse large B‐cell lymphoma cell lines SUDHL4, SUDHL6, and Farage were obtained from the American Type Culture Collection (ATCC, Manassas, VA, USA).

    Techniques: Virus, Infection, Derivative Assay, Gene Expression, Reverse Transcription, Quantitative RT-PCR, Western Blot, Expressing